College of Medicine
Han Lab
Led by Sung Min Han · Physiology and Aging
What we study
The Han Lab studies how neurons maintain function and mitochondrial health across lifespans, using the nematode C. elegans. They explore mitochondrial dynamics, stress responses, and aging impacts.
What working here is like
- Wet lab
- Worms (C. elegans)
- Coding helps
- Projects
- 3
- Papers
- 5
Current projects
Exploring the Systemic Regulation of Organismal Longevity and Health through Individual Neuron Mitochondrial Stress
Investigating how mitochondrial stress in neurons affects overall organismal aging and health.
The role and regulation of mitochondrial localization in mature neurons
Studying how mitochondria position themselves in neurons and their role in aging.
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Investigating the Neurotoxic Effects of 6PPD Exposure in a C. elegans Model of Parkinson's Disease
Scientists expose a tiny worm model of Parkinson’s disease to a tire chemical, 6PPD, to see if it worsens nerve damage. They measure nerve cell health, movement, and oxidative stress to determine the chemical’s impact.
From the Spring 2025 UF Undergraduate Research Symposium · abstract book, p. 114
Summaries are written by AI in plain language from public symposium abstracts and may be imperfect.
Recent papers, explained
- Mitochondrial Signaling and Stress Responses, Key Regulators of Aging and Longevity
2026 · BioEssays
Reviews how mitochondria influence aging and longevity through stress responses.
- Exploring age-related iron dysregulation: effects on longevity, body size, and behavior in C. elegans
2025 · Experimental Gerontology
Studies how iron changes with age in worms, affecting lifespan and movement.
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- 6-PPD induces mitochondrial dysfunction and reduces healthspan and lifespan through SKN-1 in Caenorhabditis elegans
2025 · Journal of Hazardous Materials
Finds 6-PPD harms worms by damaging mitochondria and reducing lifespan.
- Bisphenol TMC disturbs mitochondrial activity and biogenesis, reducing lifespan and healthspan in the nematode Caenorhabditis elegans
2025 · Ecotoxicology and Environmental Safety
Shows bisphenol TMC shortens worm lifespan and causes developmental delays.
- Inhalation exposure to a tire-derived pollutant (6-PPD) triggers lung injury and mitochondrial dysfunction in mice
2025 · Ecotoxicology and Environmental Safety
Shows tire chemical 6-PPD damages mouse lungs and causes inflammation.
Plain-language summaries are AI-written and reviewed by us; check the paper for details.
How to reach out
Sung Min Han hasn't set up this page yet. Their UF Scholars profile lists their publications.